It is not for direct human consumption or medical use
Figure 5 In the context of ALD, ethanol and its primary metabolite, acetaldehyde, induce redistribution of TJ proteins to disrupt apical junctional complexes, facilitating increased intestinal permeability (34, 110115)
Excessive production of ECM components, coupled with chronic inflammation, promotes the progression from steatosis to hepatic fibrosis, cirrhosis, and ultimately liver failure ( Although MAFLD provides a broader and more inclusive diagnostic framework, current pharmacological strategies are largely based on evidence from NASH and liver fibrosis trials, which remain applicable in the context of progressive MAFLD
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