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Therefore, while the concept of exercise-mediated inhibition of cuproptosis is compelling, further mechanistic and longitudinal studies are needed to clarify causality and optimize exercise prescriptions for atherosclerosis prevention through this pathway

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The relative potency at each receptor target has been described in early preclinical and clinical publications, with retatrutide demonstrating particularly strong GLP-1 and GIP receptor activity alongside moderate glucagon receptor activation, though precise potency ratios remain subject to ongoing characterisation

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