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1 Introduction Myocarditis is a heterogeneous disease characterized by myocardial inflammation and cardiomyocyte injury ( The natural course of myocarditis exhibits a distinct stage-dependent progression ( Within this pathological context, alterations in cardiac energy metabolism have attracted increasing attention ( Experimental studies have consistently demonstrated that cardiomyocytes in myocarditis exhibit a metabolic shift from oxidative phosphorylation toward increased reliance on glycolysis, even under relatively oxygen-replete conditions ( In addition to cardiomyocytes, infiltrating immune cells within the myocardium undergo substantial metabolic remodeling

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