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Neuroinflammation Neuroinflammation is increasingly recognized as a key factor interacting with neurobiological correlates of the neuropsychiatric long COVID syndrome, such as brain serotonin depletion [111], hypothalamicpituitaryadrenal (HPA) axis dysregulation [57], and impaired hippocampal neurogenesis and plasticity [13, 61]

The 600% increase in GLP-1 usage from 2019 to 2024 reflects both the medications' effectiveness and the urgent need for obesity treatments ^18

Liver glutathione declines 35-50% in aged animals [7] , while brain glutathione drops by at least 50% over adult aging [8]

Typical Maintenance Dose Ranges Across GLP-1 Medications Standard maintenance doses vary widely across available GLP-1 formulations