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(2020) specifically addressed NSAID-induced gut damage a clinically relevant injury model and documented that BPC-157 administration stabilized the claudin-4 and occludin tight junction proteins that are degraded by NSAID exposure, providing a mechanistic explanation for the peptides permeability-protective effects

In this paper we develop a mathematical model for APAP metabolism that allows us to study, in silico , how various doses of APAP are metabolized and whether or not a dose exceeds the capacity of the liver to synthesize sufficient GSH

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