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This review is significant as it provides a comprehensive exploration of the role of inflammasomes in APAP-induced liver injury, a critical but often underemphasized aspect of the pathophysiology of this condition

It is possible that compensatory mechanisms could develop that reduce efficacy with prolonged use

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For example, in PDAC, PD-1 signaling has been shown to enhance tumor progression via the MAPK and PI3K/AKT pathways [89]