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During hepatic regeneration, increased activity of urokinase plasminogen activator (uPA) triggers extracellular matrix (ECM) remodeling involving cascades of metalloproteinase activation which lead to the release of active HGF in the ambient environment of hepatocytes and in circulation and this is followed by a plethora of events to restore the architecture of the liver 41,42

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The execution of ferroptosis involves complex regulation of multiple metabolic pathways (Figure 1), including disruptions in iron homeostasis, abnormalities in amino acid antioxidant systems, and the accumulation of lipid peroxides (Zheng et al., 2025

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