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The phenotype is thought to arise due to deficits in cerebral energy production during and after fetal development, the accumulation of the potentially neurotoxic guanidinoacetate and other guanidino compounds, and possible abnormalities in ongoing neural signaling associated with abnormalities in the creatinephosphocreatine system (88)
Research Evidence Preclinical research on BPC-157 spans tendon and ligament repair, inflammatory bowel disease models, gastric ulcer healing, bone repair, and neurological injury
Deepa G, MHSc (Diab), Dip (Diab)
USA 107 , 1600916012 (2010)
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