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Epub 2021/09/07

pneumoniae , not only in the wt , but also in the gpoA, tpxD , and ahpD mutants, we used the same infection model for the A549 pneumocytes and RAW 264.7 macrophages, but with a treatment during infection of 5 mM NAC for 3 h for A549 cells, and 10 mM NAC for 1 h for RAW 264.7 cells

Beyond the impact of endothelial damage, peritubular capillary rarefaction, pericyte detachment, nephron loss, interstitial hypoxia, and persistent inflammation, metabolic disturbances in TECsparticularly defective FAO and lipid accumulationhave been identified as key contributors to EMT and the progression of renal fibrosis through maladaptive repair mechanisms (Miguel et al., 2021)