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Coptis chinensis , disrupts Candida albicans cell-wall architecture by increasing surface exposure of -glucans and chitin, thereby weakening the barrier and heightening susceptibility to immune attack and antifungal agents (Shi et al., 2017

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Understanding how these pathways complement each other provides the mechanistic rationale for combining CJC-1295 No DAC (a GHRH analog) with Ipamorelin (a selective GHRP)

Its unique mechanism of action ensures it remains a staple in regenerative medicine