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Angiogenesis, wound-repair, and preclinical systemic effects In preclinical models, GHK-Cu promotes angiogenesis (new blood-vessel formation), attracts repair cells such as macrophages and capillary cells to injury sites, and accelerates wound healing across a range of tissues and species skin, hair follicles, gastrointestinal lining, bone, and more

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Substances that are absorbed through the buccal route go directly into the systemic circulation and this eliminates disadvantages imposed by oral intake, such as the decreased ability of GSH to pass through the gastrointestinal tract as an intact molecule

The most powerful tools in our arsenal today are GLP-1 receptor agonists like Semaglutide and Tirzepatide