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Another strategy is to destroy the primary defense, i.e., bacteria's cell wall, through novel targets, different from the target of the commonly used -lactam antibiotics [74, 75]

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These effects mirror those observed with exercise, leading to interest in ERR agonists as potential "exercise mimetics." The compound also increases mitochondrial respiration in vitro, consistent with the role of ERRs in regulating mitochondrial function

Several hypertension models suggest a close association between decreased NO bioavailability and increased oxidative stress and hypertension