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However, further studies are necessary to understand the physiopathological mechanisms implicated (Schetters et al., 2018) because it is unknown whether OS is a primary cause that induces the initiation of neurodegenerative diseases or if it is a side effect associated with the spread of damage to nerve cells (Jenner, 2003

(2020) reported elevated levels of Cyanobacteria and Proteobacteria in SPS exposed male rats

Experimental investigations suggest that it may interact with telomerase activity and influence melatonin production

327 One major mechanism of apoptosis resistance in CSCs is the upregulation of antiapoptotic proteins, including BCL-2, BCL-XL, and survivin, which inhibit the intrinsic apoptosis pathway and prevent CSC death

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