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DOI: 10.1159/000358623 [Google Scholar] Wei JP, Yang CL, Leng WH, Ding LL, Zhao GH

It inhibits NOTCH1 signaling by using its GCT activity to cleave a -glutamyl-cysteine bond at glutamate 1669 of the immature NOTCH receptor, thereby preventing the essential S1 cleavage and subsequent activation ( FIGURE 6 The physiological importance of this mechanism is underscored by severe developmental phenotypes upon its disruption

Glucose-derived ages enhance human gastric cancer metastasis through Rage/Erk/Sp1/Mmp2 cascade

But perhaps the best proof of B12s superpowers is what happens when it is not adequately present in the body

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