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Reactive oxygen species-induced actin glutathionylation controls actin dynamics in neutrophils
Further studies are necessary to determine optimal dosing regimens, long-term safety, and the precise mechanisms through which GLP-1RAs produce these analgesic effects
This review systematically introduces the antioxidant mechanism of NAC, analyzes the advancements in NAC-related research involving mesenchymal stem cells, precursor cells, innate immune cells and animal models, discusses its function using the classic oral microenvironment as an example, and places particular emphasis on the innovative applications of NAC-modified tissue engineering biomaterials
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