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This advantage is attributed, in part, to fasting-induced elevations in GH, which stimulate lipolysis and limit proteolysis during energy deficit periods [21]

L., Bonvin, A

CD36 is considered a biomarker for atherosclerotic lesion progression since it stimulates inflammatory response and foam cell formation

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We also found metabolic pathways that were uniformly altered irrespective of location such as the fatty acid degradation pathway (Fig 1C yellow nodes and Table C S1 File)