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Despite relatively high prevalence of both conditions and their debilitating impact on life (for example, one-quarter of ME/CFS patients are bed- or house-bound) [6], the understanding of mechanisms leading to the development of fatigue is poor

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Presently, there is no evidence available from studies in humans to support or refute this theory

Beyond structural and sensory-motor roles, numerical studies have indicated a propulsive function for them as well, presenting the hypothesis that chemical energy released by a rapidly disassembling microtubule can be converted into useful work needed to generate the large mechanical forces that propel chromosomes during cell replication [133]