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Excessive generation of reactive oxygen species activates redox-sensitive signalling pathways, including NF-B, leading to sustained release of pro-inflammatory cytokines such as TNF-, IL-6, and IL-1, which impair fibroblast function, collagen synthesis, and neovascularisation

Our findings challenge the notion that GLP-1R agonistmediated improvements in glucose metabolism are solely driven by reduced food intake and highlight the importance of developmental timing and nutritional context when evaluating safety and efficacy of GLP-1 receptor agonists

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Serotonin metabolism is dysregulated in cholangiocarcinoma, which has implications for tumor growth

, BPC-157 , TUDCA