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Long term safety data from GLP-1 agonist medications now extends beyond five years for some patients

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An increase in lactate output by brain tissue serves to meet the energy needs of glutamate-activated neurons

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LC was found to protect against glutamate neurotoxicity mediated by increasing the activation of metabolic receptors

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If GLP-1 drugs are indeed curbing alcohol consumption, the impact could be profound

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Finally, we reviewed current therapeutic strategies targeting ferroptosis in diabetic wounds, which have shown promising effects in in vitro and in vivo studies

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