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Ferroptosis requires acyl-CoA synthetase long-chain family member 4 (ACSL4)-mediated membrane enrichment of the -6 PUFA arachidonic acid (AA), which is prone to oxidation 2,6,7

Mutations in Gli3 result in a shortened cell cycle, impaired cortical lamination, and cortical neuron formation and are responsible for various morphological CNS abnormalities, such as Greig cephalopolysyndactyly syndrome (GCPS), Pallister-Hall syndrome (PHS) and polydactyly and syndactyly (178)

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