At the same time it serves conversely as a signaling molecule to activate a rescue/defense system for restoring the redox homeostasis in plant cells
Several animal studies have further shown that administration of GLP-1RAs, before or shortly after experimentally-induced stroke, increased angiogenesis, neurogenesis and cerebral blood flow (CBF), reduced neuroinflammation, oxidative stress, excitotoxicity, BBB leakage and apoptosis, and induced dose-dependent decreases in infarct volume [60,61,62]
Others argue that systemic administration is just as effective and much simpler
Attenuated GLP1 secretion in obesity: Cause or consequence
A patient's genetic profile does not dictate which medication will work, but it can help inform a more informed discussion with a healthcare provider about personalized dosing or compound selection