However, cancer cells exhibit a heightened reliance on de novo fatty acid synthesis to sustain their lipid metabolic equilibrium and establish diverse functional lipid reservoirs, which are essential for the initiation, survival, and progression of HCC(Hepatocellular carcinoma) ( Research suggests that the transition from lipid uptake to de novo lipogenesis in cancer cells leads to an increase in membrane lipid saturation, resulting in higher levels of saturated and monounsaturated phospholipids, thereby reducing lipid peroxidation potential to potentially protect cancer cells from oxidative damage ( de novo fatty acid (FA) synthesis, the citrate carrier (CIC/SLC25A1) transports TCA cycle product citrate across the mitochondrial inner membrane into the cytoplasm to participate in metabolism ( in vivo was effectively suppressed by PTL, demonstrating no significant observed toxic reactions

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O.et al (2012)
Thereby, BPC 157 is effective in wound healing much like it is effective in counteracting bleeding disorders, produced by amputation, and/or anticoagulants application