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Unlike full-length VEGF, QK is a smaller and more stable molecule that retains the ability to bind endothelial receptors (particularly VEGFR-1), activating key biological pathways involved in vascular growth

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Normally, NF-B is bound to its inhibitor IkB, thus remaining confined to the cytoplasm: to be activated NF-B is phosphorylated by the IB kinase (IKK) complex, which leads to the IkB degradation and allows the transcription factor translocation into the nucleus [117]

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