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Much evidence suggests that activation of anorexogenic proopiomelanocortin (POMC) neurons and simultaneous inhibition of orexigenic neuropeptide Y/Agouti-related peptide (NPY/AgRP) neurons in the ARC appears to be one of the major mechanisms for GLP-1R mediated inhibition of food intake

Acknowledgements This work is supported by DST/SERB-SRG file no

GLP-1 secretion assays are shown as three experimental data points or more, with each experimental data point representing the mean of three technical repeats, unless otherwise indicated in the legends to the figures

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