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Primary hypothyroidism in an adult patient with protein-calorie malnutrition: a study of its mechanism and the effect of amino acid deficiency

In many in vivo disease studies or oxidative stress stimulation models, GPX1 deficiency enhances cell damage, apoptosis, and even leads to cell death

Mechanisms controlling cellular and systemic iron homeostasis

For example, NOX inhibitors effectively suppress ROS production, thereby reducing oxidative stress and protecting cells from oxidative damage

Additionally, EDT increased the levels of Fe 2+ and the production of ROS while lowering the protein levels of SLC7A11 and GPX4