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Differences in experimental design are also likely to contribute to differences in results

Transient stress or increased cytoplasmic TDP-43 triggers phase separation into toxic cytoplasmic droplets that block nuclear import, deplete nuclear TDP-43, and cause cell death, offering a present mechanistic model for ALS/FTD pathology (Gasset-Rosa et al., 2019)

To assay the ability of HPV-16 E7 Mut to interact with GSTP1 in vivo , control, HPV-16 E7-infected and HPV-16 E7 Mut-infected HaCaT cells were lysed and immunoprecipitation using an anti-HA antibody was performed

To solve this, we developed a way to wrap glutathione in "liposomes": tiny bubbles made of the same building blocks that make up our cell walls

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