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Through co-immunoprecipitation (CO-IP) experiments, we discovered that FOXO4-DRI prevents the binding of FOXO4 to P53, facilitating the phosphorylated P53 nuclear exclusion, which subsequently trigger BAX and cleaved caspase-3, leading to the apoptosis of senescent cells

Emerging studies suggest that instead of directly modulating neurotransmitter levels, brain iron overload could indirectly influence neurotransmission through epigenetic mechanisms (Ye et al., 2019

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