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1 Introduction Kidney disease is a major global non-communicable disease (NCD), with a global prevalence exceeding 10% [affecting ~850 million people when including acute kidney injury (AKI), kidney failure, and dialysis/transplant recipients] ( Cell death is a finely regulated process that occurs through various molecular pathways ( Recent studies have confirmed that the kidney is particularly susceptible to redox imbalance, and ferroptosis plays a significant role in the pathophysiology of various kidney diseases, emerging as a new research hotspot in the field of renal fibrosis ( Figure 1
Nile grass rats ( Arvicanthis niloticus ), a diurnal rodent, exposed to short photoperiod (5 h light/19 h dark) for 6 weeks presented reduced saccharin preference, higher immobility time in the forced swim test, and no changes on time spent in the light side of the dark/light box in comparison to controls (Figure 1), rats develop into depressive-like phenotypes (i.e., anhedonia, sexual dysfunction, increased immobility in the forced swim test) ( Landgraf et al
Key mechanistic observations from laboratory studies: VEGF upregulation: BPC-157 is thought to upregulate VEGF expression
doi: 10.3389/fmicb.2022.983358 213
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