Although both antioxidants used as pre-treatment (Mel and GSH) mitigated the harmful effects of CARB, Mel appeared to exert comparatively stronger inhibitory effects on apoptotic (Cas-3) and inflammatory (NF-B, TNF-, IL-6) responses
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RUVBL1/2 Blockade Targets YTHDF1 activity to suppress m6A-dependent oncogenic translation and colorectal tumorigenesis
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