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The glucose-dependent mechanism of insulin release distinguishes GLP-1 agonists from many other antidiabetic medications, resulting in a lower risk of hypoglycemia when used as monotherapy

They stimulate the release of insulin from the pancreas, suppress glucagon secretion (which helps reduce blood sugar levels), and promote feelings of fullness, leading to reduced food intake and potential weight loss

Reconstitution (if applicable) Some compounded GLP-1 medications arrive as lyophilized powder that requires reconstitution with bacteriostatic water

Visceral fat produces more aromatase, more inflammatory cytokines, and more adipokines that impair sexual function

Provider assessment of these markers guides dose adjustments and treatment optimization