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If optimization does not produce results, combination therapy should be considered

BPC-157 activates angiogenic repair pathways, TB-500 enhances actin polymerization and cellular migration for tissue remodeling, and GHK-Cu stimulates collagen and elastin synthesis critical for venous wall structural integrity

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Microbiome-targeted therapies, such as fecal microbiota transplantation, antibiotics, bile acid modulators, and probiotics, demonstrate potential in combating intestinal dysbiosis, reducing bacterial populations responsible for endotoxin production, decreasing gut barrier permeability, restoring normal bile acid metabolism, improving liver chemistries, and eventually mitigating disease progression [36]

Visually, funnel plots did not demonstrate publication bias