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Pyke, C

The development of hepatic steatosis is likely due to blockade of glucagons lipolytic properties in the liver, therefore making GCGR antagonism an untenable strategy

Journal of Clinical Pharmacology, 63(12), 1121-1130

However, despite mucosal GSH depletion, there was no evidence of mucosal injury as revealed by the lack of histologic changes of samples from DEM-treated rats

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