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Moreover, in 1-antitrypsin deficiency mice models with inhibited host hepatocytes proliferation, wild-type donor hepatocytes replaced 20%98% of host hepatocytes (Ding et al., 2011)
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This is further supported by scRNA-seq results from our study, which show that many DEGs associated with immunity, cellular senescence, degenerative disease, or inflammation were significantly upregulated or downregulated in the model group but restored toward baseline in the WCV-treated group
