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This rapid degradation is the primary reason that GLP-1 receptor agonist drugs require structural modifications such as DPP-4 resistance mutations, albumin binding, or Fc fusion to achieve a pharmacologically useful duration of action
AMPs) and activates myeloid-derived suppressor cells (MDSCs), suppressing immune function
These findings suggest that even in a weight-reduced state from untreated obesity, the DVC amylin and GLP-1 receptors continue to be a viable drug target for sustained weight loss
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