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used HTS strategy to identify a small molecule that blocks CagA (a part of the T4SS apparatus, containing an NTP binding motif), and the molecule prevented CagA from translocation into AGS cells

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Hence, while treatments like chemotherapy, radiation, and immunotherapy initially showed promise, the emergence of resistant tumor cells decreased therapeutic effectiveness

Potential Benefits Chromatin activation (in-vitro): The peptide is reported to decondense chromatin and de-repress silenced genes in elderly lymphocytes an unproven mechanistic hypothesis, not a clinical benefit [5] [6]