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In white adipose tissue of obese, insulin-resistant and aged mammals, NNMT is dramatically upregulated and because it consumes both the universal methyl donor (SAM) and the NAD precursor (nicotinamide), its hyperactivity simultaneously depletes the cellular methylation reserve and the NAD-salvage pool [1,2]

It plays an important role in cellular energy production and the maintenance of keratin infrastructure

B12 is crucial for ATP production, supporting increased stamina, focus, and mental clarity