Additionally, radiation-induced ROS comprises not only peroxides but an array of other ROS such as hydroxyl, peroxyl, superoxide, and hydroperoxyl, among others, which add more complexity to the mechanism of radiation injury [36, 37]
One of the explanation is that c-MET-positive lymphoma cells have retained the physiological growth control by c-MET and possessed high proliferative index, Ki67, resulting in cell cycle progression and possibly chemotherapy sensitivity [20]
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It has also been reported that pulmonary function is negatively related to oxidative stress markers and positively related to serum antioxidant levels (10)
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