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HMGB1 regulates erastin-induced ferroptosis via RAS-JNK/p38 signaling in HL-60/NRASQ61L cells

Figures were conducted using GraphPad Prism 8.0.2 (GraphPad Software, San Diego, CA, USA)

This structural change is reversible and involves the formation of an intramolecular disulfide bond and a non-covalent dimer

If successful, the class action could result in compensation for affected individuals and serve as a catalyst for increased transparency in pharmaceutical marketing practices

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