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Indeed, increased levels of proinflammatory cytokines, including IL-6, IL-1, IL-17A, and IL-22, are observed in psoriasis, leading to excessive ROS generation from both endogenous and exogenous sources, resulting in increased oxidative stress

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These effects are accompanied by reduced accumulation of reactive oxygen species and improved maintenance of cellular homeostasis throughout the lifespan

Oxidative modification of low-density lipoprotein (LDL) cholesterol forms oxidized LDL (oxLDL), which is highly atherogenic [68]

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