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In addition, although statistical significance could not be calculated for values below detection limits, acetyl-CoA and ATP levels can be considered to have increased upon L-carnitine administration, which suggests that oxidation of lipids was enhanced to produce acetyl-CoA, resulting in ATP production (Table 1)

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This article compares the two as research compounds: what each one is, the mechanisms each is investigated for, how their preclinical research profiles differ, and why they are sometimes examined together