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It is conceivable that this general mechanism can explain Mercury-induced oxidative stress not only through a continuous depletion of the reduced glutathione pool, but also by interfering with enzymes operating through active sites based on vicinal thiol groups or which are regulated by glutathionylation of sensing cysteine residues

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Second, IGF-1 targets should be benchmarked against Tanner-stage-adjusted reference ranges rather than chronological age alone, because a 14-year-old at Tanner stage IV has a substantially different IGF-1 reference interval than a 14-year-old at Tanner stage II [7]

125 The expression of the antioxidant enzymes in islet cells is low, leading to a greater tendency for them to be affected by oxidative stress