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Novel mechanism of inhibition of rat kidney-type glutaminase by bis-2-(5-phenylacetamido-1,2,4-thiadiazol-2-yl)ethyl sulfide (BPTES)

Such a mechanism may explain the peptide's ability to enhance healing without causing excessive or dysregulated tissue growth, as its effects remain dependent on physiological growth factor signaling that is naturally upregulated during injury and repair processes

The study on mice found that lowering wound size led to increased epithelial division

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Research also notes increased IFN- and IL-2 with reduced IL-6 and IL-10 levels