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penneri alone was unable to consume BB or produce TMA, whereas only E

Our findings that diet and particularly meat consumption does not associate with increased TMAO levels in our population do not contradict a number of well-designed human interventional trials 9,10,11 that have established a clear link between meat intake or L -carnitine supplementation and TMAO circulating levels

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Our recent work further illustrated that glycolysis-related genes are upregulated or downregulated during low-fat diet or high-fat diet feeding, respectively 51