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Dihydrofolate reductase deficiency due to a homozygous dhfr mutation causes megaloblastic anemia and cerebral folate deficiency leading to severe neurologic disease

Diminishing results despite consistent dosing is another signal

Key finding: AOD 9604 increased bone formation rate by 67%, increased lumbar spine BMD by 12%, and increased trabecular density

The peptides mechanism appears to preferentially mobilize fat from metabolically active depots while preserving muscle tissue, creating favorable body recomposition rather than non-selective weight loss

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