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However, glucagon also: Increases energy expenditure: Glucagon promotes thermogenesis and fat oxidation Targets the liver directly: The liver has no GLP-1 or GIP receptors but is rich in glucagon receptors Reduces liver fat: Glucagon receptor activation may ease oxidative stress in liver mitochondria Provides anti-fibrotic benefits: Improved fat oxidation can improve mitochondrial function When combined with GLP-1 and GIP agonism, the glucagon components blood sugar effects appear to be counterbalanced while its metabolic benefits are retained
Do not share your semaglutide pen or needles with anyone
Evidence suggests that DPP-4 inhibitors reduce urinary albumin excretion, oxidative stress, and renal dysfunction in patients with T2D independent of their glucose-regulatory activity [117, 118]
REFERENCES ABOUT THIS ARTICLE Cite this article: Wang Y., Feng Z
