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Male patients predominantly experienced AEs within the initial 30-day window following treatment initiation, whereas female patients exhibited a delayed onset pattern, with the majority of AEs emerging after 360 days of continuous therapy

Acute ischaemic brain injury disrupts physiological brainheart signalling through the autonomic nervous system, the hypothalamicpituitaryadrenal axis and the immune system, potentially leading to heart damage and dysfunction (the so-called strokeheart syndrome)

This is a direct consequence of its forced-turnover mechanism

Pancreatitis incidence remains rare and statistically comparable between both GLP-1 medications when prescribed appropriately and monitored by healthcare providers