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The presence of oxidative stress in the periphery in secondary progressive MS (SPMS) patients, 7 in whom the disease is thought to be more neurodegenerative and confined to the CNS, 1 supports the hypothesis that oxidative stress is part of the pathogenesis and not merely a result of tissue destruction

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However, in a studying employing the human-like SCID mouse model, melatonin induced regression of transplanted human endometriotic lesions (166)