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Moreover, we define specific AH subpopulations of hepatocytes with inflammatory and hepatobiliary cell profile, scar-associated macrophages and pro-fibrogenic endothelial cells, probably involved in the pathogenesis of AH
Activation of hepatic stellar cells (HSCs) is one of the major events responsible for fibrotic progression, whereas liver sinusoidal endothelial cells (LSECs) play a crucial role in inhibiting the activity of HSCs and stimulating immune response
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On the day of semaglutide injection, estrous cycle phase was determined for each mouse (Fig