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The Epithalon effect is therefore understood not as a direct building block of telomeres, but as a signal that switches a normally repressed gene back on

Mitochondrial transfer mediated by mesenchymal stem cells (MSCs) could restore the mitochondrial pool of neurons and inhibit ferroptosis, 951 providing a promising clinical translational strategy for ferroptosis-related central nervous system diseases based on stem cell-mediated mitochondrial therapy

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