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It is also strongly pro-angiogenic: in rat and endothelial-cell models it promoted new blood-vessel formation and blood-flow recovery by up-regulating the VEGFR2AkteNOS pathway[2]

This bidirectionality may explain why some nutrient interventions fail [e.g., vitamin B12 supplementation is ineffective in patients with gut microbiota dysbiosis (149)]

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It modulates immune responses, promoting the efficient elimination of pathogens and foreign invaders

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